Microangiopathic hemolytic anemia: Difference between revisions
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***Congenital ([[Upshaw-Shulman syndrome|Upshaw-Shulman]]) |
***Congenital ([[Upshaw-Shulman syndrome|Upshaw-Shulman]]) |
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***Acquired |
***Acquired |
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| − | **[[Hemolytic |
+ | **[[Hemolytic-uremic syndrome]] (HUS) |
***Typical (Shiga-producing [[Escherichia coli]]) |
***Typical (Shiga-producing [[Escherichia coli]]) |
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***[[Atypical hemolytic uremic syndrome]] (aHUS) (congenital complement-overactivation) |
***[[Atypical hemolytic uremic syndrome]] (aHUS) (congenital complement-overactivation) |
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Latest revision as of 12:07, 10 May 2023
Differential Diagnosis
- Primary/thrombotic microangiopathies (TMA)
- Thrombotic thrombocytopenic purpura (TTP)
- Congenital (Upshaw-Shulman)
- Acquired
- Hemolytic-uremic syndrome (HUS)
- Typical (Shiga-producing Escherichia coli)
- Atypical hemolytic uremic syndrome (aHUS) (congenital complement-overactivation)
- Thrombotic thrombocytopenic purpura (TTP)
- Secondary
- Antiphospholid antibody syndrome
- Malignant hypertension
- Lymphomas and solid tumours
- Scleroderma crisis
- Drug-associated: calcineurin inhibitors, quinine, ticlopidine
- Vasculitis
- Infections: HIV, influenza, EBV, parvovirus
- Disseminated intravascular coagulation (DIC)
- HELLP syndrome
