Antibiotic-associated diarrhea: Difference between revisions
From IDWiki
(Created page with "== Differential Diagnosis == * Clostridioides difficile * Other microbes: Clostridium perfringens type A (food poisoning), Staphylococcus aureus, Salmonella ent...") |
No edit summary |
||
| Line 1: | Line 1: | ||
== |
==Differential Diagnosis== |
||
* |
*[[Clostridioides difficile]] |
||
* |
*Other microbes: [[Clostridium perfringens]] type A (food poisoning), [[Staphylococcus aureus]], [[Salmonella enterica]], and possible [[Candida albicans]] |
||
* |
*Direct effects on intestinal tract |
||
** |
**[[Erythromycin]] and [[clavulanate]] cause increased motility |
||
** |
**[[Penicillins]] may rarely cause a [[segmental colitis]] |
||
* |
*Effects on the fecal microbiota |
||
** |
**Decreased carbohydrate metabolism, leading to osmotic diarrhea |
||
** |
**Breakdown of primary bile acids, leading to secretory diarrhea |
||
* |
*Concomitant use of laxatives, antacids, contrast, lactose- or sorbitol-containing products, NSAIDs, antiarrhythmic drugs, and cholinergic drugs |
||
[[Category:Adverse drug reactions]] |
[[Category:Adverse drug reactions]] |
||
Revision as of 18:13, 17 September 2020
Differential Diagnosis
- Clostridioides difficile
- Other microbes: Clostridium perfringens type A (food poisoning), Staphylococcus aureus, Salmonella enterica, and possible Candida albicans
- Direct effects on intestinal tract
- Erythromycin and clavulanate cause increased motility
- Penicillins may rarely cause a segmental colitis
- Effects on the fecal microbiota
- Decreased carbohydrate metabolism, leading to osmotic diarrhea
- Breakdown of primary bile acids, leading to secretory diarrhea
- Concomitant use of laxatives, antacids, contrast, lactose- or sorbitol-containing products, NSAIDs, antiarrhythmic drugs, and cholinergic drugs
References
- ^ Sara Blaabjerg, Daniel Artzi, Rune Aabenhus. Probiotics for the Prevention of Antibiotic-Associated Diarrhea in Outpatients—A Systematic Review and Meta-Analysis. Antibiotics. 2017;6(4):21. doi:10.3390/antibiotics6040021.
