Aconite poisoning
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Revision as of 13:59, 30 August 2022 by Aidan (talk | contribs) (Created page with "* Toxic alkaloid derived from monkshood, wolfsbane, and other members of the genus Aconitum * Bins and opens sodium channels, preventing their inactivation * Manifestations include GI (nausea, vomiting, cramping), neurologic (numbness, particularly of face and mouth then progressing to limbs, muscle weakness, and coma), and cardiac (hypotension, arrhythmias) * Management is supportive ** Bradycardia: atropine ** Long QT/TdP: magniseium ** Arrhythmias: cardioversion ** EC...")
- Toxic alkaloid derived from monkshood, wolfsbane, and other members of the genus Aconitum
- Bins and opens sodium channels, preventing their inactivation
- Manifestations include GI (nausea, vomiting, cramping), neurologic (numbness, particularly of face and mouth then progressing to limbs, muscle weakness, and coma), and cardiac (hypotension, arrhythmias)
- Management is supportive
- Bradycardia: atropine
- Long QT/TdP: magniseium
- Arrhythmias: cardioversion
- ECMO
