Streptococcus bovis group: Difference between revisions
From IDWiki
Streptococcus bovis group
mNo edit summary |
No edit summary |
||
Line 9: | Line 9: | ||
***Biotype II/2: β-glucuronidase positive |
***Biotype II/2: β-glucuronidase positive |
||
*Members include: |
*Members include: |
||
− | **[[Streptococcus equinus |
+ | **[[Streptococcus equinus]] |
− | **[[Streptococcus |
+ | **[[Streptococcus lutetiensis]] (previously [[Streptococcus infantarius]] subsp. ''coli'') (biotype II/1) |
− | **[[Streptococcus infantarius |
+ | **[[Streptococcus infantarius]] subsp. ''infantarius'' (biotype II/1) |
− | **[[Streptococcus alactolyticus |
+ | **[[Streptococcus alactolyticus]] |
− | **[[Streptococcus gallolyticus |
+ | **[[Streptococcus gallolyticus]] subsp. ''gallolyticus'' (biotype I), classically associated with [[colon cancer]] and [[infective endocarditis]] |
− | **[[Streptococcus gallolyticus |
+ | **[[Streptococcus gallolyticus]] subsp. ''pasteurianus'' (biotype II/2) |
− | **[[Streptococcus gallolyticus |
+ | **[[Streptococcus gallolyticus]] subsp. ''macedonicus'' |
==Further Reading== |
==Further Reading== |
Latest revision as of 15:53, 16 September 2024
Background
Microbiology
- Essentially, the Streptococcus bovis group consists of bacteria are non-enterococcal Group D streptococci
- Historically grouped by biotype:
- Biotype I: ferments mannitol
- Biotype II: cannot ferment mannitol
- Biotype II/1: β-glucuronidase negative
- Biotype II/2: β-glucuronidase positive
- Members include:
- Streptococcus equinus
- Streptococcus lutetiensis (previously Streptococcus infantarius subsp. coli) (biotype II/1)
- Streptococcus infantarius subsp. infantarius (biotype II/1)
- Streptococcus alactolyticus
- Streptococcus gallolyticus subsp. gallolyticus (biotype I), classically associated with colon cancer and infective endocarditis
- Streptococcus gallolyticus subsp. pasteurianus (biotype II/2)
- Streptococcus gallolyticus subsp. macedonicus
Further Reading
- An update on the Streptococcus bovis group: classification, identification, and disease associations. J Clin Microbiol. 54:1694 –1699. doi: 10.1128/JCM.02977-15.