Paroxysmal sympathetic hyperactivity: Difference between revisions
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* 80% of patients with PSH have traumatic brain injury |
* 80% of patients with PSH have traumatic brain injury |
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==Clinical |
==Clinical Manifestations== |
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* Episodes of increased sympathetic output triggered by external stimulation |
* Episodes of increased sympathetic output triggered by external stimulation |
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* Symptoms most commonly include [[Causes::tachycardia]], [[Causes::diaphoresis]], and increased motor activity |
* Symptoms most commonly include [[Causes::tachycardia]], [[Causes::diaphoresis]], and increased motor activity |
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Latest revision as of 02:19, 22 July 2020
Background
Pathophysiology
- Thought to be damage to inhibitory sympathetic centres of the diencephalon
- This allows spinal cord stimuli to be amplify through uninhibited positive feedback
Epidemiology
- 80% of patients with PSH have traumatic brain injury
Clinical Manifestations
- Episodes of increased sympathetic output triggered by external stimulation
- Symptoms most commonly include tachycardia, diaphoresis, and increased motor activity
- Other symptoms include fever (up to 70%), hypertension, and tachypnea
Management
- Treated with beta blockers, especially propanolol
- May add clonidine for better blood pressure control
