Paroxysmal sympathetic hyperactivity: Difference between revisions

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m (Text replacement - "Clinical Presentation" to "Clinical Manifestations")
 
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* 80% of patients with PSH have traumatic brain injury
* 80% of patients with PSH have traumatic brain injury


==Clinical Presentation==
==Clinical Manifestations==
* Episodes of increased sympathetic output triggered by external stimulation
* Episodes of increased sympathetic output triggered by external stimulation
* Symptoms most commonly include [[Causes::tachycardia]], [[Causes::diaphoresis]], and increased motor activity
* Symptoms most commonly include [[Causes::tachycardia]], [[Causes::diaphoresis]], and increased motor activity

Latest revision as of 02:19, 22 July 2020

Background

Pathophysiology

  • Thought to be damage to inhibitory sympathetic centres of the diencephalon
  • This allows spinal cord stimuli to be amplify through uninhibited positive feedback

Epidemiology

  • 80% of patients with PSH have traumatic brain injury

Clinical Manifestations

Management

  • Treated with beta blockers, especially propanolol
  • May add clonidine for better blood pressure control